What is the difference between Citrate, Gluconate, and Picolinate derivatives of vitamins?

Is there a difference and if so, what is it?

I enjoy a zinc deficiency and I enjoy been prescribed to thieve a Zinc supplement.

Is there a difference between the three different kind of Zinc?

If there is, please explain what it is and how it affects the digestion of Zinc, the concentration of Zinc, etc.

Thank you for help!

Most appreciatively,

I hold no clues. Try wikipedia
The difference is in bioavailability (how much of the vitamin brackish that is absorbed). Gluconate salt of vitamins are more bioavailable than Citrate. I don't know about Picolinate.
move about to the next knit:
visit pubmed.org to read abstract

pubmed likes citrate

My thought is they adjectives make the zinc available but the nifty item is are they metabolized at different rates If you take a zinc tablet once per year one of the three will have the more gradual adjectives day forceful dose.

zinc picolinate attaches to the most common plasma protein albumin, thus it might be a more gradual dose form than gluconate which attaches zinc to glucose to build it absorbable. On the spooooooky side zink picolinate is insulin mimetic, less insulin response is associated near longevity.

Citrate looks like the process to go as it is more significant at raising zinc containing enzymes

pubmed.org say
[Effect of different concentrations of various zinc complexes (picolinate, citrate, 8-hydroxyquinolate) surrounded by comparison with sulfate on zinc supply status within rats][Article in German]
Roth HP, Kirchgessner M.
12 groups of 8 young-looking male Sprague-Dawley rats received a semisynthetic casein-diet, whose zinc concentration (1.3 ppm) be adjusted to 5, 10 and 15 ppm by supplementation of an assortment of Zn complexes or salts resembling Zn citrate, Zn picolinate, Zn 8-hydroxyquinolate and Zn sulfate. After 24 days all animals be killed and examined on parameter of zinc supply status. The weight gain of the animals beside 5 ppm dietary zinc was strongly reduced compared to groups near the higher dietary zinc content. The type of the supplemented Zn compound showed contained by no way an influence on the live-weight of the animals. The zinc concentration of the tissues, too, be only dependent on the stratum of the dietary zinc concentration and not on the type of supplemented zinc compound. The activity of the alkaline phosphatase within serum, a zinc metalloenzyme, was altogether reduced within the zinc-deficient animals with 5 ppm dietary zinc content, but showed a significant greater activity surrounded by the citrate and 8-hydroxyquinolate group than in the sulfate and picolinate group. Also the percent zinc-binding size of serum, a good indicator for estimating the zinc supply status of rats, showed a better zinc supply of the citrate group next to 5 as well as 10 ppm dietary zinc, compared near the other groups on the same dietary zinc content. The serum zinc concentration of rats beside 5 ppm as Zn citrate was more than twice highly developed than in animals given zinc as picolinate, 8-hydroxyquinolate or sulfate. The results indicate a better utilization of zinc chelated by citric sour than by picolinic acid, 8-hydroxyquinoline or as saline like sulfate. But the superior bioavailability of zinc in human milk should not solely be attributed to the presence of citrate.

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